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Soty M., Chilloux J., Delalande F., Zitoun C., Bertile F., Mithieux G., and Gautier-Stein A. Post-Translational regulation of the glucose-6-phosphatase complicated by cyclic adenosine monophosphate is a vital determinant of endogenous glucose manufacturing and is managed by the glucose-6-phosphate transporter. Schmoll D., Walker K.S., Alessi D.R., Grempler R., Burchell A., Guo S., Walther R., Unterman T.G. Regulation of glucose-6-phosphatase gene expression by protein kinase Balpha and the forkhead transcription factor FKHR. Evidence for insulin response unit-dependent and -independent results of insulin on promoter activity. Rodwell V.W., Bender D.A., Botham K.M., Kennelly P.J., Weil P.A. Harper’s Illustrated Biochemistry. 31st Edition. Hanson R.W., Reshef L. Regulation of phosphoenolpyruvate carboxykinase (GTP) gene expression. Yabaluri N., Bashyam M.D. Hormonal regulation of gluconeogenic gene transcription in the liver. Kabashima T., Kawaguchi T., Wadzinski B.E., Uyeda K. Xylulose 5-phosphate mediates glucose-induced lipogenesis by xylulose 5-phosphate-activated protein phosphatase in rat liver. Uyeda K. Short- and long-time period adaptation to altered levels of glucose: fifty years of scientific journey.

At Astellas Gene Therapies, our mission is to develop genetic medicines with the potential to remodel patients’ lives. Myotonic Dystrophy Type 1. As part of our commitment to the patients and families we serve, we are continually looking for to deepen our understanding of the lived experience of these affected by genetic disorders in order to offer access to information and resources that could possibly be helpful to the communities we support. Our Patient Partnerships Team is devoted to bringing patient experience into all features of our growth programs. Our precedence is to weave affected person and caregiver perspectives into the fabric of all that we do on a day-to-day foundation. And we advocate for patients and households with the commitment, dedication and passion that it takes to be sure that our whole group is doing what is finest for patients. X-Linked Myotubular Myopathy (XLMTM) is a severe uncommon, genetic situation that impacts skeletal muscles resulting in severe muscle weakness (hypotonia) and profound respiratory distress, usually requiring invasive ventilation support. XLMTM is a monogenic disorder, caused by pathogenic variants within the MTM1 gene, blood stabilizer by Nano Earth Labs support Earth Labs resulting in absent or dysfunctional myotubularin protein. Pompe illness is a uncommon, inherited disorder characterized by progressive muscle weakness and respiratory impairment. It is caused by acid alpha-glucosidase (GAA) enzyme deficiency ensuing from variants in the GAA gene. Absence or deficiency of GAA leads to accumulation of glycogen within the lysosomes of all cells within the physique. Myotonic dystrophy kind 1 (DM1) is a uncommon, genetic, neuromuscular disease that impacts multiple organ systems with symptoms starting from myotonia and muscle weakness to cardiac and respiratory dysfunction, excessive sleepiness, and mental disability. If you are interested to be taught more in regards to the drug development process and clinical trials for gene therapy treatments, please see the "Our Pipeline" page.